The first section considers selected aspects of molecular mechanisms, including selectivity of toxic agents and repair processes in the nervous system, toxicity of oxygen, fibers and aflatoxins.
Mechanisms of Toxicity. WithFrancesco De Matteis, Lewis L. Smith. chapter Chapter 116 pages
The first section considers selected aspects of molecular mechanisms, including selectivity of toxic agents and repair processes in the nervous system, toxicity of oxygen, fibers and aflatoxins. Mechanisms of Toxicity. chapter Chapter 116 pages. Aspects of Selectivity and Mechanisms in Neurotoxicity.
Table of contents (11 chapters). Cellular Specific Toxicity in the Lung. Smith, Lewis L. (et a.
eBook 62,99 €. price for Russian Federation (gross). Table of contents (11 chapters).
Lewis L. Smith, Francesco De Matteis. The first section considers selected aspects of molecular mechanisms, including selectivity of toxic agents and repair processes in the nervous system, toxicity of oxygen, fibers and aflatoxins. The second section discusses the interactions of carcinogens with DNA, and other targets, and their relevance to both molecular dosimetry of exposure and development of cancer.
The first section considers selected aspects of molecular mechanisms, including selectivity of toxic agents and repair processes in the nervous system, toxicity of oxygen, fibers and aflatoxins. Medical Science Nonfiction.
Ian N. H. White, Francesco De Matteis, +5 authors David H Phillips. Potentiation of the cell specific toxicity of paraquat by 1,3-bis(2-chloroethyl)-1-nitrosourea (BCNU)
Highly Influential Citations56. Ian N. Chronic administration of tamoxifen to female rats causes hepatocellular carcinomas. Potentiation of the cell specific toxicity of paraquat by 1,3-bis(2-chloroethyl)-1-nitrosourea (BCNU). Implications for the heterogeneous distribution of glutathione (GSH) in rat lung. Simon J. Hardwick, Arlette Adam, Lewis L. Smith, Gerald M. Cohen. Biochemical pharmacology.
Francesco De Matteis. Heme is vital to our aerobic universe. Heme cellular content is finely tuned through an exquisite control of synthesis and degradation. Heme deficiency is deleterious to cells, whereas excess heme is toxic. Francesco De Matteis. We have measured liver heme oxygenase, a heat shock protein known to be increased under conditions of oxidative stress, to obtain additional evidence for an oxidative stress mechanism in hepatic uroporphyria induced by hexachlorobenzene (HCB)
Are you Francesco De Matteis? Register this Author. Food Chem Toxicol 2004 May;42(5):785-90
Are you Francesco De Matteis? Register this Author. Register with ORCID iD. PUBLICATIONS 11. About publications (11) network. Department of Cellular and Molecular Pharmacology, The Liver Center, University of California, San Francisco, 94158, USA. View Article. Food Chem Toxicol 2004 May;42(5):785-90. Department of Anatomy, Pharmacology and Forensic Medicine, Medical Pharmacology Section, Via Pietro Giuria 13, 10125 Torino, Italy.
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